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Regarding treatment strategies, it is suggested that the marked decrease in GPx1 expression may be the major mechanism of tumor sensitization to anthracyclines [93] and that GPx1 can partially inhibit doxorubicin-induced cell death-related signaling in breast cancer by interfering with the activation of the sphingomyelin-ceramide pathway [85]

[DOI] [PMC free article] [PubMed] [Google Scholar] 45.Chen J., Berry M.J

The interest in tendon recovery is backed by preclinical work such as this study examining how BPC-157 influences tendon fibroblast healing and growth factor expression in injured connective tissue

The resulting linear peptide undergoes self-assembly into nanofibers within the cell