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3.7.3 Mechanisms of association between UPF intake and ASD There have been a range of recent studies that have shown that high dietary intake of UPF may contribute to an increased risk of developing ASD, which may be mediated through a range of different pathophysiological mechanisms, including lipid dysregulation, oxidative stress and neuroinflammation (Table 2)

This has resulted in the emergence of compounded formulations of these drugs, which produce entities that bypass regulatory measures